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             morbidity: scope, clinical presentation and treatment options.   tion perpetuated by modern diet as a promoter of obesity and
             Brain Inj. 2014;28:261–270.                        osteoporosis. Arh Hig Rada Toksikol. 2014;65:139–148.
          23.  Ruff RM, Iverson GL, Barth JT, et al. Recommendations for   41.  Minshall D. Gulf War Syndrome: a review of current knowledge
             diagnosing a mild traumatic brain injury: a National Acad-  and understanding. J R Nav Med Serv. 2014;100:252–258.
             emy of Neuropsychology education paper. Arch Clin Neuro-  42.  Veitch DP, Friedl KE, Weiner MW. Military risk factors for
             psychol. 2009;24:3–10.                             cognitive decline, dementia and Alzheimer’s disease. Curr Al-
          24.  Maas AI, Harrison-Felix CL, Menon D, et al. Common data   zheimer Res. 2013;10:907–930.
             elements for traumatic brain injury: recommendations from   43.  Hermes G, Ajioka JW, Kelly KA, et al. Neurological and be-
             the interagency working group on demographics and clinical   havioral abnormalities, ventricular dilatation, altered cellular
             assessment. Arch Phys Med Rehabil. 2010;91:1641–1649.  functions, inflammation, and neuronal injury in brains of
          25.  Saatman KE, Duhaime AC, Bullock R, et al. Classification of   mice due to common, persistent, parasitic infection. J Neuro-
             traumatic brain injury for targeted therapies. J Neurotrauma.   inflammation. 2008;5:48.
             2008;25:719–738.                                44.  Teeling JL, Perry VH. Systemic infection and inflammation in
          26.  Reger MA, Gahm GA, Swanson RD, et al. Association be-  acute CNS injury and chronic neurodegeneration: underlying
             tween  number of  deployments  to  Iraq  and  mental  health   mechanisms. Neuroscience. 2009;158:1062–1073.
             screening outcomes in US Army soldiers. J Clin Psychiatry.
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          27.  Gronwall D, Wrightson P. Cumulative effect of concussion.
             Lancet. 1975;306:995–997.
          28.  Guskiewicz KM, McCrea M, Marshall SW, et al. Cumula-  Dr Figueroa is cofounder and president of the Brain Health
             tive  effects  associated  with  recurrent  concussion  in  colle-  & Healing Foundation and former director of Scientific Re-
             giate football players: the NCAA Concussion Study. JAMA.   search at the Restorix Research Institute. Dr Figueroa has
             2003;290:2549–2555.                             been performing neurological clinical research since 1995 in
          29.  Richmond E, Rogol AD. Traumatic brain injury: endocrine   the field of Alzheimer’s research, as well as basic research in
             consequences in children and adults. Endocrine. 2014;45:3–8.  neuron biology, cancer research, bioengineering, and the bio-
          30.  Pekic S, Popovic V. Alternative causes of hypopituitarism:   physics of water in cells. Dr Figueroa received his doctoral de-
             traumatic  brain  injury,  cranial  irradiation,  and  infections.   gree in 2003 from the University of Washington’s Program in
             In: Eric Fliers MK, Johannes AR, eds.  Handbook of clini-  Neurobiology & Behavior in the Department of Environmen-
             cal neurology. Amsterdam, The Netherlands: Elsevier; 2014:   tal Health and Toxicology. His doctoral training was followed
             271–290.
          31.  Berg C, Oeffner A, Schumm-Draeger PM, et al. Prevalence of   up by two postdoctoral fellowships within the Department
             anterior pituitary dysfunction in patients following traumatic   of Bioengineering (2003–2008).He was a former director of
             brain injury in a German multi-centre screening program.   scientific research for clinical trials using hyperbaric oxygen
             Exp Clin Endocrinol Diabetes. 2010;118:139–144.  therapy to treat traumatic brain injuries in Veterans and active
          32.  Samadani  U, Reyes-Moreno  I,  Buchfelder  M.  Endocrine   duty Servicemembers. Dr Figueroa has published widely in the
             dysfunction following traumatic brain injury: mechanisms,   areas of neurobiology, bioengineering, evolutionary biology,
             pathophysiology and clinical correlations.  Acta Neurochir   and clinical research involving hyperbaric medicine. E-mail:
             Suppl. 2005;93:121–125.                         bhif.figueroa@gmail.com.
          33.  Desai A, Kevala K, Kim HY. Depletion of brain docosahexae-
             noic acid impairs recovery from traumatic brain injury. PLoS   Dr Wright has served in a number of capacities in his career
             One. 2014;9:e86472.
          34.  Pu H, Guo Y, Zhang W, et al. Omega-3 polyunsaturated fatty   as an Air Force officer, flight surgeon, educator, and researcher.
             acid supplementation improves neurologic recovery and at-  His most recent role has been as an investigator in a national,
             tenuates white matter injury after experimental traumatic   multisite clinical trial using hyperbaric oxygen for traumatic
             brain injury. J Cereb Blood Flow Metab. 2013;33:1474–1484.  brain injury. Dr Wright is a 1973 graduate from the University
          35.  Lewis M, Ghassemi P, Hibbeln J. Therapeutic use of omega-3   of Chicago and completed surgical and plastic surgery residen-
             fatty acids in severe head trauma. Am J Emerg Med. 2013;31:   cies at the University of Chicago (1974–77) and UCLA (1977–
             273 e5–8.                                       79), respectively. He has completed fellowships as a Clinical
          36.  Hasadsri L, Wang BH, Lee JV, et al. Omega-3 fatty acids as a   and Research Fellow (Burns) at Harvard Medical School in the
             putative treatment for traumatic brain injury. J Neurotrauma.   Shriners’ Burn Institute, Boston (1975) and Hyperbaric Medi-
             2013;30:897–906.
          37.  Paterniti I, Impellizzeri D, Di Paola R, et al. Docosahexae-  cine at Brooks Air Force Base, Texas (1999–2000). Dr Wright
             noic acid attenuates the early inflammatory response follow-  was commissioned into the Air Force in 1982 (Captain) and
             ing spinal cord injury in mice: in-vivo and in-vitro studies. J   served in several capacities. He retired 1 February 2010 (Colo-
             Neuroinflammation. 2014;11:6.                   nel) as the 720th Special Tactics Group Surgeon. Dr Wright
          38.  Hall JC, Priestley JV, Perry VH, et al. Docosahexaenoic acid,   has published widely in the areas of surgery, plastic surgery,
             but not eicosapentaenoic acid, reduces the early inflammatory   and hyperbaric medicine, and has supervised clinical research
             response following compression spinal cord injury in the rat.   in the areas of predictors of success in Special Tactics selection
             J Neurochem. 2012;121:738–750.                  and training of personnel.
          39.  Bazan NG, Molina MF, Gordon WC. Docosahexaenoic acid
             signalolipidomics in nutrition: significance in aging, neuroin-
             flammation, macular degeneration, Alzheimer’s, and other neu-
             rodegenerative diseases. Annu Rev Nutr. 2011;31:321–351.






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